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Hannah Garner 1 , Karin E de Visser 2 3 Affiliations 1 Division of Tumour Biology & Immunology, Oncode Institute, Netherlands Cancer Institute, Amsterdam, Netherlands.. Leiden University Medical Centre, Leiden, Netherlands. [email protected]. PMID: 32024984 DOI: 10.1038/s41577-019-0271-z Abstract.
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Karin E. de Visser Nature Reviews Cancer 16 , 431-446 ( 2016) Cite this article 34k Accesses 1045 Citations 21 Altmetric Metrics Key Points In patients with solid cancers, neutrophils expand.
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Karina Elizabeth de Visser (Karin de Visser, 1975), [1] also named "Karin E. de Visser", is a researcher at the Netherlands Cancer Institute. Her research considers metastatic formation and how the immune system influences how people respond to cancer treatment. Early life and education
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Karin de Visser @deVisserKarin Tumor Immunologist @NKI_nl @oncodeinstitute interested in crosstalk between the immune system and breast cancer, metastasis, myeloid and adaptive immune cells Amsterdam, The Netherlandsnki.nl/research/resea… Joined December 2022 40Following 397Followers Tweets Tweets & replies Media Likes Karin de Visser's Tweets
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Karin E. de Visser Nature Reviews Immunology 20 , 483-497 ( 2020) Cite this article 20k Accesses 205 Citations 108 Altmetric Metrics Abstract Metastatic disease is responsible for.
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Seth B Coffelt 1 , Max D Wellenstein 1 , Karin E de Visser 1 Affiliation 1 Division of Immunology, Netherlands Cancer Institute, Plesmanlaan 121, 1066 CX Amsterdam, The Netherlands. PMID: 27282249 DOI: 10.1038/nrc.2016.52 Abstract Neutrophils are indispensable antagonists of microbial infection and facilitators of wound healing..
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Prof. dr. Karin E. de Visser is group leader at the Division of Tumor Biology & Immunology at the Netherlands Cancer Institute, alongside her appointment as group leader at Oncode Institute and as full professor of Experimental Immunobiology of Cancer at Leiden University. The overall goal of her research is to dissect how the immune system influences metastatic breast cancer.
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Karin E. de Visser's research while affiliated with Netherlands Cancer Institute and other places Overview What is this page? This page lists the scientific contributions of an author, who.
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Karin de Visser Inflammation and cancer Metastasis formation and unresponsiveness to conventional therapies are the challenges in cancer patient care that urgently need solutions. The overall goal of our research is to gain novel mechanistic insights into how the immune system influences these processes, with a primary focus on breast cancer.
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KARIN DE VISSER OUR RESOURCES Resources. Our team has developed a preclinical mouse model of invasive lobular breast cancer metastasis. We used this model to unravel mammary tumor-induced pro-metastatic inflammation (IL-17-producing γδ T cells and.
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KARIN DE VISSER Karin de Visser Group leader +31 20 512 6104 [email protected] Prof. dr. Karin E. de Visser obtained her PhD at the Division of Immunology at the Netherlands Cancer Institute in Amsterdam in the field of tumor immunotherapy.
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Karin E de Visser Netherlands Cancer Institute · Division of Immunology PhD Connect with experts in your field Join ResearchGate to contact this researcher and connect with your scientific.
Oratie Karin de Visser ‘Het immuunsysteem is een tweesnijdend zwaard’
In February Karin de Visser received a VICI grant from NWO to investigate how genetic changes in breast cancer cells influence the immune system of patients. De Visser's research group is also affiliated with the Oncode Institute.
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KARIN DE VISSER OUR RESEARCH Inflammation and cancer. Metastasis formation and unresponsiveness to conventional therapies are the challenges in cancer patient care that urgently need solutions. The overall goal of our research is to gain novel mechanistic insights into how the immune system influences these processes, with a primary focus on.
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Karin E. de Visser Johanna A. Joyce DOI: https://doi.org/10.1016/j.ccell.2023.02.016 The evolving tumor microenvironment: From cancer initiation to metastatic outgrowth Summary Cancers represent complex ecosystems comprising tumor cells and a multitude of non-cancerous cells, embedded in an altered extracellular matrix.
